EUROKERATOCONUS 2007

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ag2005

Dear colleagues, dear friends,


Though not recent, the keratoconus will be in the ophthalmic limelight on November 30th 2007 during the first European congress on keratoconus. The treatment of this disease – a disease which nowadays remains the first indicated cause for corneal transplantation among young adults – is at the beginning of a major turning point in its evolution. That’s why I am glad to coordinate the organisation of this first congress with Pr Joseph Colin. I am delighted at the collaboration of the CRK with the Association Kératocône to organise a meeting for patients on that day.

I think it is important, first and foremost, to mention that all these advances have been made possible thanks to a better knowledge of the innermost structure of this mysterious dystrophy. With this aim in view, the numerous histological works, particularly Professor Pouliquen’s, have really brought about the “modern” keratoconus era. The advance of para clinical exploration initially, and most importantly of the topography of the cornea, have allowed to make more and more precise detection of its formes frustes, which are real nightmares for the refractive surgeon. More recent technologies (aberrometry, Scheimpflug rotating camera, genetic studies will no doubt cast a new light on this difficult diagnosis to the point of abnormality.

Contactology has also advanced, with lenses increasingly better fitting lenses that can be adapted to the unusual fitting to the unusual curvatures of keratoconus, offering remarkable safety, quality of vision and comfort. It has allowed to postpone still further for as long as possible the surgical operation which is often dreaded by our young patients. However, though still concerning 20% of these patients, the surgical technique of transplantation has considerably evolved too, thanks to the rapid expansion of lamellar transplantations which today allow to keep the patient’s endothelium and thus lessen the risk of rejection as much as possible. Besides short of obtaining an etiological the treatment of the keratoconus for now thanks to the combined advances of genetic therapy and molecular biology, new preserving strategies are developing, such as corneal collagen cross-linking which could soon become a precious tool in the fight against this dystrophy.

We have finally been eager to devote this first congress to post-LASIK ectasia which is a particular but connected entity, and which won’t fail to (will undoubtedly) interest all those who practise this kind of surgery.
As you can see, the keratoconus is now an ailment which is the target of many new kinds of diagnostic and therapeutic techniques, which create wonderful hope for all our patients. Hoping this meeting will give an overview as comprehensive as possible of this fascinating but nonetheless dreadful disease, I will be seeing you in Toulouse on November 30th!

Pr François MALECAZE - CRK Toulouse  

 

ag2005Dear friends,

Post-LASIK corneal ectasias are a severe, but rare, complication of modern refractive surgery. Some risk factors are well defined, such as a corneal topographic asymmetry, a thin cornea <5000µ or a stromal residual bedding < 250µ. The existence of a family keratoconus is also an unfavourable factor. However some cases of ectasias have been observed without any known risk factor, and conversely some patients showing a risk have been treated with LASIK without developing an ectasia. This first European congress will allow to share our respective experiences on keratoconus and post-surgery ectasias.


Pr Joseph COLIN - CRK Bordeaux   



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